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Through specific alterations, adult cells can be reprogrammed to express embryonic genes, allowing them to differentiate into tissues other than their specific lineage. Efficient and scalable purification of cardiomyocytes from human embryonic and induced pluripotent stem cells by VCAM1 surface expression. Cardiac fibroblast-derived extracellular matrix biomatrix as a model for the studies of cardiac primitive cell biological properties in normal and pathological adult human heart. The applications of these new technologies are not yet fully established as part of the management of cardiac diseases but will become so in the near future. Diversity in the origins of proteostasis networks—a driver for protein function in evolution. Cell-based cardiac pumps and tissue-engineered ventricles. Aging is a biologic process that unfolds over a lifetime.

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The Intersection of Aging Biology and the Pathobiology of Lung Diseases: A Joint NHLBI/NIA Workshop

Encouragingly, animal models have shown that stem cell therapy is safe and effective. Accelerated ageing of the lung in COPD: Biofilm formation by coagulase-negative staphylococci: By far the most extensively studied subgroup is the lantibiotics belonging to class I bacteriocins. Find articles by Moises Selman.

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This was the first time that such percutaneous delivery of ECM using a transendocardial approach was carried out. Mature biofilms on stainless steel and polypropylene recalcitrant to nisin. Quantitative reconstruction of leukocyte subsets using DNA methylation. The main improvements in the early and late groups were an estimated 3. Liu Q, Wang H, editors.
Recent advances in natural product-based anti-biofilm approaches to control infections. Advances in data analysis, storage, manipulation, and access will be required to fully realize the potential of these rich resources. Sub-lethal concentrations of the lantibiotic nisin and the class II bacteriocin bovicin HC5 have also been shown to disrupt S. Enterococcus faecalis bacteriocin EntV inhibits hyphal morphogenesis, biofilm formation, and virulence of Candida albicans. Inhibition of glucan biofilm synthesis. Decellularization of living tissue using microwave chemical process for tissue-engineered scaffold applications. Genetic defects in telomerase and telomere genes markedly increase the risk of IPF and is in fact the most commonly identifiable cause of familial pulmonary fibrosis accounting for at least one-third of cases, and is a constitutional finding in at least half of IPF patients
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